Pinned Tweet
TICK tock. Your Immune clock is ticking!
-Immune Age- @OmniscopeAI is launching a suite of Immune Tracking products at #MWC2025.
-Immune Age- leverages T cells as a living sensors to sense and memorize systemic challenges. Pathogen exposure, for instance, triggers an immune lasting imprint on the T cell repertoire. However, we found that the signals encoded in T cell receptor (#TCR) sequences extend far beyond infections. They carry quantifiable insights into stress, internal and external load, and, ultimately, biological aging.
@OmniscopeAI, together with @FCBarcelona_es, now applies the potential of T cell sequencing as a Diagnostic tool to define Health, to optimize Performance and to avoid Injuries. Our T Cell Clock is a dynamic measure to quantify the accumulation of a player's load and the success of targeted interventions.
No doubt, our -Immune Age- application will go beyond the area of sports, to better define health and to extend healthy aging (#Healthspan). Our T cell sensor approach provides a new Biomarker strategy to quantify #BiologicalAge, complementing the more static Methylation clocks (@EricTopol : erictopol.substack.com/p/steve-horvat…).
Excited to see -Immune Age- to be applied in additional health intervention studies. Please reach out to collaborate! #HealthyAging #Longevity
-Immune Age- @OmniscopeAI is launching a suite of Immune Tracking products at #MWC2025.
-Immune Age- leverages T cells as a living sensors to sense and memorize systemic challenges. Pathogen exposure, for instance, triggers an immune lasting imprint on the T cell repertoire. However, we found that the signals encoded in T cell receptor (#TCR) sequences extend far beyond infections. They carry quantifiable insights into stress, internal and external load, and, ultimately, biological aging.
@OmniscopeAI, together with @FCBarcelona_es, now applies the potential of T cell sequencing as a Diagnostic tool to define Health, to optimize Performance and to avoid Injuries. Our T Cell Clock is a dynamic measure to quantify the accumulation of a player's load and the success of targeted interventions.
No doubt, our -Immune Age- application will go beyond the area of sports, to better define health and to extend healthy aging (#Healthspan). Our T cell sensor approach provides a new Biomarker strategy to quantify #BiologicalAge, complementing the more static Methylation clocks (@EricTopol : erictopol.substack.com/p/steve-horvat…).
Excited to see -Immune Age- to be applied in additional health intervention studies. Please reach out to collaborate! #HealthyAging #Longevity
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The missing link: Human-Data-In-The-Loop!
"We are still far from teaching AI how human biology really works. No AI model has yet captured the full complexity of human biology, largely because we still lack sufficient human data in the loop. Experimental model systems, while indispensable, capture only a fraction of the cellular diversity and biological variation that define human health and disease.”
The real breakthrough will come when we bring enough human data into the loop, capturing the natural perturbations of biology across individuals, ages, environments, and disease states. Only then will we be able to build comprehensive models that move beyond describing biology to reliably predicting it.
This is exactly why our work at the @humancellatlas and @OmniscopeAI is so important - Cracking the Code of Biology.
With much more human data is when the ultimate potential of AI × Biology will be realized: when predictive models generate tangible advances in human health and, ultimately, save lives.
Check out the full article at @GENbio by @julemieux1:
genengnews.com/topics/omics/s…
"We are still far from teaching AI how human biology really works. No AI model has yet captured the full complexity of human biology, largely because we still lack sufficient human data in the loop. Experimental model systems, while indispensable, capture only a fraction of the cellular diversity and biological variation that define human health and disease.”
The real breakthrough will come when we bring enough human data into the loop, capturing the natural perturbations of biology across individuals, ages, environments, and disease states. Only then will we be able to build comprehensive models that move beyond describing biology to reliably predicting it.
This is exactly why our work at the @humancellatlas and @OmniscopeAI is so important - Cracking the Code of Biology.
With much more human data is when the ultimate potential of AI × Biology will be realized: when predictive models generate tangible advances in human health and, ultimately, save lives.
Check out the full article at @GENbio by @julemieux1:
genengnews.com/topics/omics/s…
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Holger Heyn retweeted
Our warmest congratulations to #ICREAResearcher @hoheyn (CNAG) on his appointment as Vice Co-Chair of the @humancellatlas! 🧬
We wish you every success in this new role helping shape the technological future of this international initiative! 👏
We wish you every success in this new role helping shape the technological future of this international initiative! 👏
✨ Honored to step into the role of Vice Co-Chair of the Human Cell Atlas ✨ Working together with our new co-chairs Chloé Villani and Muzz Haniffa, alongside my fellow Vice Co-Chair Shyam Prabhakar.
I can’t imagine a better next leadership team for the @humancellatlas. Beyond rd, it’s a pleasure to work with such thoughtful and collaborative colleagues. This is a particularly meaningful moment for me, having been part of the HCA journey for almost a decade. I have had the privilege of co-chairing the HCA Technology Working Group and Industry Partnership Program, and more recently serving on the HCA 2.0 Task Force, helping shape the vision and strategy for this next phase.
As we move towards HCA 2.0, we are defining the future vision and roadmap for the next generation of the Human Cell Atlas #HCANext10. In my new role, I will specifically focus on leading the HCA technology roadmap, working closely with our technology community as well as biotech and pharma partners to integrate the latest single-cell and spatial technologies into the HCA roadmap. I’m very grateful to the HCA community for the trust. Excited for the next chapter of the Human Cell Atlas and for what we can build together.
@sangerinstitute @broadinstitute @ASTARsg @cnag_eu
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Holger Heyn retweeted
The 2027 HCA General Meeting will take place in The Hague, the Netherlands, on 29 June - 1 July. Early bird registration for the #HCA2027GM is now open and runs until 28 February 2027.
For more information and to register see buff.ly/YvwAilr
For more information and to register see buff.ly/YvwAilr
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TOUR DE FORCE out now! Our Phase II SERPENTINE trial: 24 patients with longitudinal biopsies, 84 sequencing libraries, 388,196 single cells and 57,902,418 T cell clonotypes!
Massive effort with @AstraZeneca , @OmniscopeAI , @VHIO, and @cnag_eu.
Immune checkpoint inhibitors (#ICIs) have limited efficacy in #metastatic #colorectal #cancer with proficient mismatch repair and microsatellite stability (pMMR/MSS). In Serpentine, we profiled patients with dual PD-L1 and CTLA-4 blockade (ipi/nivo) by integrating paired pre-treatment and on-treatment single-cell transcriptomic and T-cell receptor (TCR) profiling with ultra-deep TCR sequencing of matched peripheral blood.
Why does immunotherapy fail in MSS colorectal cancer when LIVER metastases are present?
🔎 Our findings show that the breakdown is not systemic, but local!
1. While dual immune checkpoint inhibition successfully primes and mobilizes tumor-targeting T-cell clones to both lung and liver lesions, the metastatic microenvironment determines whether that response succeeds.
2. In LUNG metastases, these cells differentiate into productive, tumor-reactive CD8+ effectors that mount an anti-tumor response. In the LIVER, however, the immune response is derailed: suppressive myeloid populations and regulatory T cells expand, exhaustion programs fragment, and effector engagement fails to restrain tumor growth.
👀 Hence, resistance in liver metastases is not a failure of initial immune priming, but a tissue-specific misdirection of immune activation. Overcoming this barrier will require therapies that address multicellular coordination within specific organ niches.
Read the full paper here:
medrxiv.org/content/10.648…
Massive effort with @AstraZeneca , @OmniscopeAI , @VHIO, and @cnag_eu.
Immune checkpoint inhibitors (#ICIs) have limited efficacy in #metastatic #colorectal #cancer with proficient mismatch repair and microsatellite stability (pMMR/MSS). In Serpentine, we profiled patients with dual PD-L1 and CTLA-4 blockade (ipi/nivo) by integrating paired pre-treatment and on-treatment single-cell transcriptomic and T-cell receptor (TCR) profiling with ultra-deep TCR sequencing of matched peripheral blood.
Why does immunotherapy fail in MSS colorectal cancer when LIVER metastases are present?
🔎 Our findings show that the breakdown is not systemic, but local!
1. While dual immune checkpoint inhibition successfully primes and mobilizes tumor-targeting T-cell clones to both lung and liver lesions, the metastatic microenvironment determines whether that response succeeds.
2. In LUNG metastases, these cells differentiate into productive, tumor-reactive CD8+ effectors that mount an anti-tumor response. In the LIVER, however, the immune response is derailed: suppressive myeloid populations and regulatory T cells expand, exhaustion programs fragment, and effector engagement fails to restrain tumor growth.
👀 Hence, resistance in liver metastases is not a failure of initial immune priming, but a tissue-specific misdirection of immune activation. Overcoming this barrier will require therapies that address multicellular coordination within specific organ niches.
Read the full paper here:
medrxiv.org/content/10.648…
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Holger Heyn retweeted
🧬 𝗡𝗲𝘄 𝗰𝗲𝗹𝗹𝘂𝗹𝗮𝗿 𝗮𝘁𝗹𝗮𝘀 𝗼𝗳 𝘁𝗵𝗲 𝗵𝘂𝗺𝗮𝗻 𝗽𝗮𝗻𝗰𝗿𝗲𝗮𝘀
Published in @Cell_Metabolism, this single-cell map offers the most comprehensive picture to date of pancreatic cell diversity, revealing how the pancreas responds to disease
📎 cell.com/cell-metabolis…
Published in @Cell_Metabolism, this single-cell map offers the most comprehensive picture to date of pancreatic cell diversity, revealing how the pancreas responds to disease
📎 cell.com/cell-metabolis…
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✨ Honored to step into the role of Vice Co-Chair of the Human Cell Atlas ✨ Working together with our new co-chairs Chloé Villani and Muzz Haniffa, alongside my fellow Vice Co-Chair Shyam Prabhakar.
I can’t imagine a better next leadership team for the @humancellatlas. Beyond rd, it’s a pleasure to work with such thoughtful and collaborative colleagues. This is a particularly meaningful moment for me, having been part of the HCA journey for almost a decade. I have had the privilege of co-chairing the HCA Technology Working Group and Industry Partnership Program, and more recently serving on the HCA 2.0 Task Force, helping shape the vision and strategy for this next phase.
As we move towards HCA 2.0, we are defining the future vision and roadmap for the next generation of the Human Cell Atlas #HCANext10. In my new role, I will specifically focus on leading the HCA technology roadmap, working closely with our technology community as well as biotech and pharma partners to integrate the latest single-cell and spatial technologies into the HCA roadmap. I’m very grateful to the HCA community for the trust. Excited for the next chapter of the Human Cell Atlas and for what we can build together.
@sangerinstitute @broadinstitute @ASTARsg @cnag_eu
I can’t imagine a better next leadership team for the @humancellatlas. Beyond rd, it’s a pleasure to work with such thoughtful and collaborative colleagues. This is a particularly meaningful moment for me, having been part of the HCA journey for almost a decade. I have had the privilege of co-chairing the HCA Technology Working Group and Industry Partnership Program, and more recently serving on the HCA 2.0 Task Force, helping shape the vision and strategy for this next phase.
As we move towards HCA 2.0, we are defining the future vision and roadmap for the next generation of the Human Cell Atlas #HCANext10. In my new role, I will specifically focus on leading the HCA technology roadmap, working closely with our technology community as well as biotech and pharma partners to integrate the latest single-cell and spatial technologies into the HCA roadmap. I’m very grateful to the HCA community for the trust. Excited for the next chapter of the Human Cell Atlas and for what we can build together.
@sangerinstitute @broadinstitute @ASTARsg @cnag_eu
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When Treatment to #Autoimmune Diseases Fail,
Combining Targeted Therapies Offers a Solution.
NOW OUT @CellRepMed! 👀
We introduced Mitigation of Non-Response Signature (MNRS), a framework that uses longitudinal blood transcriptomic data across 6 IMIDs to systematically rank complementary (and incompatible) drug pairs.
Targeted therapies reshaped how we treat immune-mediated inflammatory diseases (IMIDs), but primary non-response remains a major therapeutic ceiling. Combining treatments is a potential solution, yet prioritizing which drug pairs to test in clinical trials out of thousands of combinations is a massive hurdle.
Key Findings:
- Data-Driven Prioritization: MNRS successfully maps complementary pairs of biologics and small molecules using longitudinal patient data.
- A Winning Pair in RA: In rheumatoid arthritis, combining anti-TNF and anti-IL-6R therapies emerged as highly complementary.
- Mechanism Discovered: Single-cell analysis localized this drug complementarity specifically to CD14+ monocytes.
- In Vivo Validation: In a collagen-induced arthritis mouse model, the anti-TNF + anti-IL-6R combination significantly outperformed monotherapy.
Huge thanks to the entire team DOCTIS Project who made this work possible!
@cnag_eu Centro Nacional de Análisis Genómico (CNAG) @OmniscopeAI @DoctisEU
🔗 Read the full open-access paper here:
cell.com/cell-reports-m…
Combining Targeted Therapies Offers a Solution.
NOW OUT @CellRepMed! 👀
We introduced Mitigation of Non-Response Signature (MNRS), a framework that uses longitudinal blood transcriptomic data across 6 IMIDs to systematically rank complementary (and incompatible) drug pairs.
Targeted therapies reshaped how we treat immune-mediated inflammatory diseases (IMIDs), but primary non-response remains a major therapeutic ceiling. Combining treatments is a potential solution, yet prioritizing which drug pairs to test in clinical trials out of thousands of combinations is a massive hurdle.
Key Findings:
- Data-Driven Prioritization: MNRS successfully maps complementary pairs of biologics and small molecules using longitudinal patient data.
- A Winning Pair in RA: In rheumatoid arthritis, combining anti-TNF and anti-IL-6R therapies emerged as highly complementary.
- Mechanism Discovered: Single-cell analysis localized this drug complementarity specifically to CD14+ monocytes.
- In Vivo Validation: In a collagen-induced arthritis mouse model, the anti-TNF + anti-IL-6R combination significantly outperformed monotherapy.
Huge thanks to the entire team DOCTIS Project who made this work possible!
@cnag_eu Centro Nacional de Análisis Genómico (CNAG) @OmniscopeAI @DoctisEU
🔗 Read the full open-access paper here:
cell.com/cell-reports-m…
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osLifetime goes Clinics 👀
Case Report: #Graves’ Orbitopathy (Thyroid Eye Disease) profiling to capture immune shifts as a patient transitioned from non-response under high-dose pulse corticosteroid therapy to clinical remission following an anti-IL-6 receptor therapy (#tocilizumab).
We performed longitudinal Immune tracking for intra-individual baselines definition and to track disease initiation, evolution, and therapeutic response. Our latest preprint shows how osLifetime provides a dynamic view of the immune system for precise patient management.
Key osLifetime insights:
- Autoimmune T-Cell Activity: We tracked dynamic T-cell clonotype expansions to quantify the strength of the autoimmunity, response and relapse.
- Immune Cell States: We captured functional cellular shifts, uncovering therapeutic resistance to steroids before clinical failure manifested.
- Actionable Immune Signatures: We delivered timely, systemic readouts far beyond classical disease-centric serology and routine blood counts.
By moving from static markers to a multimodal, longitudinal immune view, #osLifetime established a scalable framework for personalized management in Graves’ Orbitopathy and other autoimmune diseases.
📄 Read the preprint on SSRN:
papers.ssrn.com/sol3/papers.cf…
Case Report: #Graves’ Orbitopathy (Thyroid Eye Disease) profiling to capture immune shifts as a patient transitioned from non-response under high-dose pulse corticosteroid therapy to clinical remission following an anti-IL-6 receptor therapy (#tocilizumab).
We performed longitudinal Immune tracking for intra-individual baselines definition and to track disease initiation, evolution, and therapeutic response. Our latest preprint shows how osLifetime provides a dynamic view of the immune system for precise patient management.
Key osLifetime insights:
- Autoimmune T-Cell Activity: We tracked dynamic T-cell clonotype expansions to quantify the strength of the autoimmunity, response and relapse.
- Immune Cell States: We captured functional cellular shifts, uncovering therapeutic resistance to steroids before clinical failure manifested.
- Actionable Immune Signatures: We delivered timely, systemic readouts far beyond classical disease-centric serology and routine blood counts.
By moving from static markers to a multimodal, longitudinal immune view, #osLifetime established a scalable framework for personalized management in Graves’ Orbitopathy and other autoimmune diseases.
📄 Read the preprint on SSRN:
papers.ssrn.com/sol3/papers.cf…
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Holger Heyn retweeted
Human Cell Atlas at 10: A Decade of Discovery and the Road to HCA 2.0
@julemieux1 spoke with @hoheyn, @cnag_eu's ICREA professor and @OmniscopeAI co-founder about the meeting and the next 10 years. Heyn has been with the HCA since (almost) the beginning.
hubs.li/Q04tqsW_0
@julemieux1 spoke with @hoheyn, @cnag_eu's ICREA professor and @OmniscopeAI co-founder about the meeting and the next 10 years. Heyn has been with the HCA since (almost) the beginning.
hubs.li/Q04tqsW_0
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Do you want to know what the next 10 years of the @humancellatlas will look like? #HCANext10 🌍 "Modeling human biology is a hard task. To get there, we will need human data at unprecedented scale, resolution, diversity, and context."
Check out my interview with the wonderful @julemieux1 at
@GENbio to learn more about our vision for the next decade: including our #Spatial and #VirtualCell programs.
The first 10 years of HCA have given us an incredible foundation. Now, it’s time to build the next generation of the Atlas. Exciting times ahead! 🚀
Read the full interview at GEN
genengnews.com/topics/omics/h…
#HumanCellAtlas #SingleCell #SpatialBiology #VirtualCell
Check out my interview with the wonderful @julemieux1 at
@GENbio to learn more about our vision for the next decade: including our #Spatial and #VirtualCell programs.
The first 10 years of HCA have given us an incredible foundation. Now, it’s time to build the next generation of the Atlas. Exciting times ahead! 🚀
Read the full interview at GEN
genengnews.com/topics/omics/h…
#HumanCellAtlas #SingleCell #SpatialBiology #VirtualCell
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OUT NOW @Cell_Metabolism ⭐⭐⭐
The Human Pancreas Atlas (EU edition).
Profiling more than 4 million cells and nuclei from 57 donors across fetal development, adult homeostasis, and type 2 diabetes (T2D). Integrating multiple modalities: sc/snRNA-seq, snATAC-seq, VASA-seq, spatial transcriptomics (Xenium) and multiplexed proteomics (CODEX).
It takes a village to build an atlas of the human pancreas. This work has been part of the @humancellatlas project, within the EU-funded #ESPACE project. Thanks to Roland Eils and Elisabetta Mereu for the exceptional leadership. This paper is a major milestone of the Human Cell Atlas, as we move into the HCA2.0 phase, broadening the scope to include human #diversity, #age, #spatial resolution, and #disease.
I am extremely proud to have been part of this endeavor, and I’m excited to move to the next phase of the Human Cell Atlas together with our colleagues and friends across the HCA community.
@cnag_eu @CarrerasIJC and many more.
Please find the paper here:
cell.com/cell-metabolis…
The Human Pancreas Atlas (EU edition).
Profiling more than 4 million cells and nuclei from 57 donors across fetal development, adult homeostasis, and type 2 diabetes (T2D). Integrating multiple modalities: sc/snRNA-seq, snATAC-seq, VASA-seq, spatial transcriptomics (Xenium) and multiplexed proteomics (CODEX).
It takes a village to build an atlas of the human pancreas. This work has been part of the @humancellatlas project, within the EU-funded #ESPACE project. Thanks to Roland Eils and Elisabetta Mereu for the exceptional leadership. This paper is a major milestone of the Human Cell Atlas, as we move into the HCA2.0 phase, broadening the scope to include human #diversity, #age, #spatial resolution, and #disease.
I am extremely proud to have been part of this endeavor, and I’m excited to move to the next phase of the Human Cell Atlas together with our colleagues and friends across the HCA community.
@cnag_eu @CarrerasIJC and many more.
Please find the paper here:
cell.com/cell-metabolis…
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Zooming into Autoimmunity: from Micro to Macro views 🔎 #Autoimmune diseases leave a blueprint hidden inside our immune cells. Our mission is turning these cellular signals into actionable clinical precision tools, from universal solutions to the exact mechanisms driving pathology.
Our vision is captured by two recent publications:
1️⃣ The Micro Focus (@Rheumatology academic.oup.com/rheumatology/a…): Translating deep single-cell profiling directly to pediatric autoimmunity. This study unmasked the explicit rogue cells driving Juvenile Idiopathic #Arthritis (oligo JIA) and its severe extra-articular complication, #uveitis. By tracking down clonally expanded, cytotoxic CD8 T cells and transcriptionally reprogrammed MAIT cells, we proved how local joint inflammation and systemic dynamics can be mapped right down to single-cell behavior.
2️⃣ The Macro View (@NatureMedicine; nature.com/articles/s4159…): A massive, global atlas profiling 6.5 million cells across 1,047 patients and 19 different conditions. This study used explainable machine learning to map out a universal model of inflammation, proving that circulating immune cells serve as dynamic, living biomarkers to decode how systemic immune-mediated diseases function in the blood.
Blood is a highly accessible, incredibly rich #diagnostic resource. By applying advanced #immunogenomics to track rogue cellular drivers, we aren't just observing autoimmune diseases, we are mapping the exact blueprints to intercept them!
#scienceisweall @OmniscopeAI
Our vision is captured by two recent publications:
1️⃣ The Micro Focus (@Rheumatology academic.oup.com/rheumatology/a…): Translating deep single-cell profiling directly to pediatric autoimmunity. This study unmasked the explicit rogue cells driving Juvenile Idiopathic #Arthritis (oligo JIA) and its severe extra-articular complication, #uveitis. By tracking down clonally expanded, cytotoxic CD8 T cells and transcriptionally reprogrammed MAIT cells, we proved how local joint inflammation and systemic dynamics can be mapped right down to single-cell behavior.
2️⃣ The Macro View (@NatureMedicine; nature.com/articles/s4159…): A massive, global atlas profiling 6.5 million cells across 1,047 patients and 19 different conditions. This study used explainable machine learning to map out a universal model of inflammation, proving that circulating immune cells serve as dynamic, living biomarkers to decode how systemic immune-mediated diseases function in the blood.
Blood is a highly accessible, incredibly rich #diagnostic resource. By applying advanced #immunogenomics to track rogue cellular drivers, we aren't just observing autoimmune diseases, we are mapping the exact blueprints to intercept them!
#scienceisweall @OmniscopeAI
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#ThrowbackSaturday ☀️
Immune cells in blood are powerful living biomarkers for diseases. Specifically, #autoimmune diseases leave their distinct molecular fingerprints in circulating immune cells.
Hence, blood is an accessible resource, not only as a universal tool for precise patient diagnosis and personalized monitoring, but also as a window to identify autoimmune-driving pathogenic rogue cells.
In our paper published in @NatureMedicine, we leveraged the power of single-cell transcriptomics to build a comprehensive, global model of inflammation. By analyzing a massive single-cell atlas of more than 6.5 million peripheral blood mononuclear cells from 1,047 patients across 19 different diseases, we moved beyond traditional, disease-centered boundaries.
Happy weekend-reading 📒
Immune cells in blood are powerful living biomarkers for diseases. Specifically, #autoimmune diseases leave their distinct molecular fingerprints in circulating immune cells.
Hence, blood is an accessible resource, not only as a universal tool for precise patient diagnosis and personalized monitoring, but also as a window to identify autoimmune-driving pathogenic rogue cells.
In our paper published in @NatureMedicine, we leveraged the power of single-cell transcriptomics to build a comprehensive, global model of inflammation. By analyzing a massive single-cell atlas of more than 6.5 million peripheral blood mononuclear cells from 1,047 patients across 19 different diseases, we moved beyond traditional, disease-centered boundaries.
Happy weekend-reading 📒
Including data from 1,047 patients across 19 inflammatory diseases, a new atlas presents a comprehensive model of inflammation in circulating immune cells.
nature.com/articles/s4159…
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Holger Heyn retweeted
Your most valuable corporate asset isn’t in a bank. It’s the "IQ Compounding" found in your leadership’s health.
In Hong Kong for Longevity in Focus, Vijay shares how Omniscope decodes immune data to capture the Wisdom Dividend.
🤝 eu1.hubs.ly/H0sKJ_H0
#ScienceIsWeAll
In Hong Kong for Longevity in Focus, Vijay shares how Omniscope decodes immune data to capture the Wisdom Dividend.
🤝 eu1.hubs.ly/H0sKJ_H0
#ScienceIsWeAll
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Holger Heyn retweeted
Your immune age, recovery, and resilience are uniquely yours. Omniscope builds cellular avatars to model health, performance, & aging in real time.
See it live at Sports Tomorrow Congress, Barcelona | Mar 2–3, 2026:
eu1.hubs.ly/H0s6bzM0
#ScienceIsWeAll #STC26
@BarcaInnoHub
See it live at Sports Tomorrow Congress, Barcelona | Mar 2–3, 2026:
eu1.hubs.ly/H0s6bzM0
#ScienceIsWeAll #STC26
@BarcaInnoHub
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Holger Heyn retweeted
Holger Heyn @hoheyn, del Laboratorio de Genómica de Célula Única del ICREA @cnag_eu @humancellatlas:
🗨️ “Esta técnica podría permitir en un futuro próximo el diseño de tratamientos inmunológicos personalizados”
#medicinapersonalizada #enfermedades #inmunologia
🗨️ “Esta técnica podría permitir en un futuro próximo el diseño de tratamientos inmunológicos personalizados”
#medicinapersonalizada #enfermedades #inmunologia
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Holger Heyn retweeted
Health isn’t abstract. It’s cellular.
At Omniscope, we decode immune aging cell by cell, turning biology into something measurable, predictable, and actionable.
See what’s next at Sports of Tomorrow Congress, Barcelona | Mar 2–3:
🤝 omniscope.ai/contact?i_m_lo…
#ScienceIsWeAll
At Omniscope, we decode immune aging cell by cell, turning biology into something measurable, predictable, and actionable.
See what’s next at Sports of Tomorrow Congress, Barcelona | Mar 2–3:
🤝 omniscope.ai/contact?i_m_lo…
#ScienceIsWeAll
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Holger Heyn retweeted
#Health isn’t static.
osLifetime tracks immune aging, inflammation, and resilience. Integrating blood data and wearables to reveal how your body is truly responding to #Life and #Time.
Explore osLifetime ➟ eu1.hubs.ly/H0rQ4C50
#ScienceIsWeAll
osLifetime tracks immune aging, inflammation, and resilience. Integrating blood data and wearables to reveal how your body is truly responding to #Life and #Time.
Explore osLifetime ➟ eu1.hubs.ly/H0rQ4C50
#ScienceIsWeAll
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Holger Heyn retweeted
🗞️A study published in @NatureMedicine and led by #ICREAresearcher Holger Heyn (@hoheyn @cnag_eu) expands the current knowledge of inflammation biology and creates a framework for classifying inflammatory diseases.
Including data from 1,047 patients across 19 inflammatory diseases, a new atlas presents a comprehensive model of inflammation in circulating immune cells.
nature.com/articles/s4159…
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